Search results for "3-Hydroxybutyric Acid"

showing 9 items of 9 documents

Role of Haptoglobin as a Marker of Muscular Improvement in Patients with Multiple Sclerosis after Administration of Epigallocatechin Gallate and Incr…

2021

Here, we report on the role of haptoglobin (Hp), whose expression depends on the synthesis of interleukin 6 (IL-6), related to the pathogenesis of multiple sclerosis (MS), as a possible marker of muscle improvement achieved after treatment with the polyphenol epigallocatechin gallate (EGCG) and an increase in the ketone body beta-hydroxybutyrate (BHB) in the blood. After 4 months of intervention with 27 MS patients, we observed that Hp does not significantly increase, alongside a significant decrease in IL-6 and a significant increase in muscle percentage. At the same time, Hp synthesis is considerably and positively correlated with IL-6 both before and after treatment

0301 basic medicineAdultMalemedicine.medical_specialtyepigallocatechin gallateMultiple Sclerosisbeta-hydroxybutyratemuscleinterleukin 6Pilot ProjectsEpigallocatechin gallateMicrobiologyBiochemistryCatechinArticlePathogenesis03 medical and health scienceschemistry.chemical_compound0302 clinical medicineBeta hydroxybutyrateInternal medicinemedicineHumansIn patientInterleukin 6Muscle SkeletalMolecular Biologybiology3-Hydroxybutyric AcidHaptoglobinsbusiness.industryInterleukin-6Multiple sclerosisHaptoglobinfood and beveragesMiddle Agedmedicine.diseaseQR1-502haptoglobin030104 developmental biologyEndocrinologychemistrybiology.proteinKetone bodiesFemalebusiness030217 neurology & neurosurgeryBiomarkersBiomolecules
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Effect of gut-derived acetate on glucose turnover in man

1988

1. The effect of acetate absorbed from the gut on glucose turnover has been determined in four healthy subjects during both fasting and an intravenous glucose infusion by using [U-13C]glucose. 2. In the first part of the study, after an overnight fast, a tracer dose of [U-13C]glucose was infused at a constant rate along with an infusion of saline for 7 h. In the second part the saline infusion was replaced by glucose at 4.25 mg min−1 kg−1. In both studies 15 mmol of sodium acetate was given by mouth at 15 min intervals from the fourth to the sixth hour. Glucose turnover, respiratory quotient, metabolic rate and blood levels of acetate, 3-hydroxybutyrate, lactate, insulin, glucagon and gastr…

AdultDietary FiberGlycerolMalemedicine.medical_specialtymedicine.medical_treatmentHydroxybutyratesGastric Inhibitory PolypeptideAcetatesFatty Acids NonesterifiedCarbohydrate metabolismGlucagonAcetic acidchemistry.chemical_compoundGastric inhibitory polypeptideLipid oxidationInternal medicinemedicineHumansInsulinCarbon RadioisotopesAcetic Acid3-Hydroxybutyric AcidChemistryRespirationInsulinGeneral MedicineMetabolismCarbon DioxideGlucagonRespiratory quotientGlucoseEndocrinologyLactatesFemaleClinical Science
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Effect of gut-derived acetate on oral glucose tolerance in man.

1988

1. Dietary fibre has a moderating impact on glucose metabolism. To test the hypothesis that this effect of fibre may be mediated by its breakdown product acetate, oral glucose tolerance tests were carried out in healthy volunteers with and without acetate. 2. Five subjects received 50 g of glucose orally while taking either acetate (15 mmol every 15 min) by mouth or chloride as control. Oral acetate made no detectable difference to glucose tolerance or to levels of free fatty acids, 3-hydroxybutyrate, lactate, insulin, glucagon and gastric inhibitory polypeptide. 3. The 50 g dose of glucose temporarily depressed acetate levels in blood. This was probably due to an interaction between glucos…

AdultDietary FiberMalemedicine.medical_specialtyTime Factorsmedicine.medical_treatmentLumen (anatomy)HydroxybutyratesGastric Inhibitory PolypeptideXyloseCarbohydrate metabolismAcetatesFatty Acids NonesterifiedGlucagonchemistry.chemical_compoundGastric inhibitory polypeptideInternal medicinemedicine3-Hydroxybutyric AcidHumansInsulinGlucose tolerance testmedicine.diagnostic_test3-Hydroxybutyric AcidInsulinGeneral MedicineGlucose Tolerance TestGlucagonEndocrinologyGlucosechemistryLactatesFemaleClinical science (London, England : 1979)
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Metabolomics of Human Amniotic Fluid and Maternal Plasma during Normal Pregnancy

2016

Metabolic profiles of amniotic fluid and maternal blood are sources of valuable information about fetus development and can be potentially useful in diagnosis of pregnancy disorders. In this study, we applied 1H NMR-based metabolic profiling to track metabolic changes occurring in amniotic fluid (AF) and plasma (PL) of healthy mothers over the course of pregnancy. AF and PL samples were collected in the 2nd (T2) and 3rd (T3) trimester, prolonged pregnancy (PP) until time of delivery (TD). A multivariate data analysis of both biofluids reviled a metabolic switch-like transition between 2nd and 3rd trimester, which was followed by metabolic stabilization throughout the rest of pregnancy proba…

B Vitamins0301 basic medicineAmniotic fluidPhysiologyMaternal HealthPlacentalcsh:MedicineSpectrum analysis techniquesBiochemistryAcetoacetatesFetal DevelopmentPlasmachemistry.chemical_compoundGlucose MetabolismPregnancyPyruvic AcidBlood plasmaMedicine and Health SciencesMetabolitesAmino Acidslcsh:ScienceMultidisciplinary3-Hydroxybutyric AcidOrganic CompoundsObstetrics and GynecologyHematologyVitaminsKetonesBody FluidsChemistryBloodmedicine.anatomical_structurePregnancy Trimester SecondPhysical SciencesMetabolomeKetone bodiesCarbohydrate MetabolismFemaleAnatomyResearch Articlemedicine.drugPyruvateAdultmedicine.medical_specialtyPregnancy Trimester ThirdGestational AgeCholinesBiologyBlood PlasmaYoung Adult03 medical and health sciencesNMR spectroscopyInternal medicinePlacentamedicineHumansMetabolomicsCarnitineFetusPregnancy030102 biochemistry & molecular biologylcsh:ROrganic ChemistryChemical CompoundsBiology and Life SciencesAmniotic Fluidmedicine.diseaseResearch and analysis methodsMetabolismGlucose030104 developmental biologyEndocrinologychemistryWomen's Healthlcsh:QPyruvic acidAcidsPLOS ONE
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Protective action of 1,3-butanediol in cerebral ischemia. A neurologic, histologic, and metabolic study.

1987

1,3-Butanediol (BD) is converted in the body to β-hydroxybutyrate, and previous studies have shown that hyperketonemia had beneficial effects in experimental models of generalized hypoxia. The aim of this study was to determine if BD would reduce brain damage following cerebral ischemia. A transient forebrain ischemia of 30-min duration was induced by the four-vessel occlusion technique in control and BD-treated rats (25 mmol/kg, i.p.; 30 min prior to ischemia). BD treatment led to significant improvement of neurologic deficit during the 72-h recovery period and reduced neuronal damage in the striatum and cortex but not in the CA1 sector of the hippocampus. Evaluation of cerebral energy me…

Blood GlucoseMalemedicine.medical_specialtyIschemiaHydroxybutyratesBlood PressureBrain damageHippocampusPhosphocreatinechemistry.chemical_compoundInternal medicinemedicine13-ButanediolAnimalsEnergy chargeButylene GlycolsCerebral CortexNeurons3-Hydroxybutyric Acidbusiness.industryBrainHypoxia (medical)medicine.diseaseCorpus StriatumRatsEndocrinologyNeurologychemistryIschemic Attack TransientLactic acidosisKetone bodiesNeurology (clinical)medicine.symptomCardiology and Cardiovascular MedicinebusinessEnergy MetabolismJournal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism
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Fasting prior to transient cerebral ischemia reduces delayed neuronal necrosis.

1990

A transient brain ischemia of 30-min duration was induced by the four-vessel occlusion technique in normally fed and in 48-hr-fasted rats. Evaluation of brain damage 72 hr after ischemia showed that fasting reduced neuronal necrosis in the striatum, the neocortex, and the lateral part of the CA1 sector of hippocampus. Signs of status spongiosis in the pars reticulata of the substantia nigra were seen in 75% of fed rats and in only 19% of fasted rats. The protective effect was associated with reduction in mortality and in postischemic seizure incidence. The metabolic changes induced by fasting were evaluated before and during ischemia. After 30 min of four-vessel occlusion, fasted rats showe…

Blood GlucoseMalemedicine.medical_specialtyIschemiaHydroxybutyratesSubstantia nigraBlood PressureBrain damageBiochemistryBrain ischemiaCellular and Molecular Neurosciencechemistry.chemical_compoundNecrosisReference ValuesInternal medicinemedicineAnimalsNeuronsGlycogen3-Hydroxybutyric Acidbusiness.industryAdenine NucleotidesBrainRats Inbred StrainsFastingmedicine.diseaseRatsEndocrinologyGlucosechemistryIschemic Attack TransientOrgan SpecificityLactic acidosisAnesthesiaKetone bodiesLactatesNeurology (clinical)medicine.symptomPars reticulatabusinessEnergy MetabolismMetabolic brain disease
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Vibrio ponticus sp. nov., a neighbour of V fluvialis-V. furnissii clade, isolated from gilthead sea bream, mussels and seawater.

2004

A new Vibrio species, Vibrio ponticus, is proposed to accommodate four marine bacteria isolated from sea water, mussels and diseased sea bream (Sparus aurata), at the Mediterranean coast of Spain. Strains are Gram negative, slightly halophilic bacteria that require Na+ ion for growth, oxidase and catalase positive, negative for arginine dihydrolase and ornithine decarboxylase but positive for lysine decarboxylase and indole, and utilize beta-hydroxybutyrate as a sole carbon source. Phylogenetic analysis locate these marine bacteria in the vicinity of the V. fluvialis-V. furnissii clade, sharing with these two species 16S rDNA sequence similarities slightly above 97% (97.1 and 97.3%, respect…

DNA BacterialIndolesCarboxy-LyasesHydrolasesMolecular Sequence DataBiologyOrnithine DecarboxylaseApplied Microbiology and BiotechnologyMicrobiologyDNA RibosomalMicrobiologyMarine bacteriophageVibrionaceaeVibrio InfectionsRNA Ribosomal 16SSequence Homology Nucleic AcidMediterranean SeaAnimalsSeawaterRibosomal DNAEcology Evolution Behavior and SystematicsPhylogenyVibrioLysine decarboxylase3-Hydroxybutyric AcidFatty AcidsNucleic Acid HybridizationSequence Analysis DNA16S ribosomal RNAbiology.organism_classificationCatalaseVibrioSea BreamBivalviaSpainVibrio InfectionsPhenazinesGentian VioletOxidoreductasesBacteriaSystematic and applied microbiology
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Possible Reduction of Cardiac Risk after Supplementation with Epigallocatechin Gallate and Increase of Ketone Bodies in the Blood in Patients with Mu…

2020

Multiple sclerosis (MS) is a neurodegenerative disease that causes anthropometric changes characterised by functional disability, increase in fat mass, and decrease in lean mass. All these variables are related to a greater cardiac risk. The polyphenol epigallocatechin gallate (EGCG) and an increase in ketone bodies in the blood have been shown to have beneficial effects on anthropometric and biochemical variables related to cardiovascular activity. The aim of this study was to analyse the impact of the intervention with EGCG and ketone bodies on cardiac risk in MS patients. A population of 51 MS patients were randomly assigned to a control group and an intervention group (daily dose of 800…

MalePilot ProjectsKetone Bodies030204 cardiovascular system & hematologyEpigallocatechin gallatemultiple sclerosisCatechinBody Mass Indexchemistry.chemical_compound0302 clinical medicineFunctional abilityeducation.field_of_studyNutrition and Dieteticsbiology3-Hydroxybutyric AcidAnthropometryfood and beveragesMiddle AgedPON1C-Reactive ProteinTreatment OutcomeCardiovascular DiseasesKetone bodiesFemalelcsh:Nutrition. Foods and food supplyAdultmedicine.medical_specialtycardiac riskepigallocatechin gallateWaistCardiotonic AgentsMultiple SclerosisPopulationlcsh:TX341-641Article03 medical and health sciencesInternal medicinemedicineHumanseducationSerum AlbuminAnalysis of VarianceWaist-Height Ratiobusiness.industryAryldialkylphosphataseParaoxonaseEndocrinologychemistryHeart Disease Risk FactorsDietary Supplementsketone bodiesLean body massbiology.proteinbusiness030217 neurology & neurosurgeryFood Science
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Ketogenic diet in neuromuscular and neurodegenerative diseases.

2014

An increasing number of data demonstrate the utility of ketogenic diets in a variety of metabolic diseases as obesity, metabolic syndrome, and diabetes. In regard to neurological disorders, ketogenic diet is recognized as an effective treatment for pharmacoresistant epilepsy but emerging data suggests that ketogenic diet could be also useful in amyotrophic lateral sclerosis, Alzheimer, Parkinson’s disease, and some mitochondriopathies. Although these diseases have different pathogenesis and features, there are some common mechanisms that could explain the effects of ketogenic diets. These mechanisms are to provide an efficient source of energy for the treatment of certain types of neurodege…

Mitochondrial Diseasesmedicine.medical_treatmentlcsh:MedicineDiseaseReview ArticleBiologyBioinformaticsGeneral Biochemistry Genetics and Molecular BiologyAlzheimer DiseaseDiabetes mellitusmedicineHumansAmyotrophic lateral sclerosisKetogenic diet metabolic diseases preventionGeneral Immunology and Microbiology3-Hydroxybutyric AcidAmyotrophic Lateral Sclerosislcsh:RBrainParkinson DiseaseGeneral Medicinemedicine.diseaseGlycogen Storage DiseaseObesityGlucoseMitochondrial biogenesisBiochemistryAlzheimer's diseaseMetabolic syndromeDiet KetogenicKetogenic diet
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